Biblio
Modeling mutation-specific arrhythmogenic phenotypes in isogenic human iPSC-derived cardiac tissues. Sci Rep. 2024;14(1):2586.
. A disease-specific iPS cell resource for studying rare and intractable diseases. Inflamm Regen. 2023;43(1):43.
Novel Calmodulin Variant p.E46K Associated With Severe Catecholaminergic Polymorphic Ventricular Tachycardia Produces Robust Arrhythmogenicity in Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes. Circ Arrhythm Electrophysiol. 2023:e011387.
Uniform transgene activation in Tet-On systems depends on sustained rtTA expression. iScience. 2023;26(10):107685.
. Disrupted Cav1.2 Selectivity Causes Overlapping Long QT and Brugada Syndrome Phenotypes in CACNA1C-E1115K iPS Cell Model. Heart Rhythm. 2022.
The oncogene-dependent resistance to reprogramming unveils cancer therapeutic targets. Cell Rep. 2022;39(4):110721.
Capturing human trophoblast development with naive pluripotent stem cells in vitro. Cell Stem Cell. 2021.
De Novo DNA Methylation at Imprinted Loci during Reprogramming into Naive and Primed Pluripotency. Stem Cell Reports. 2019.
Inducible Transgene Expression in Human iPS Cells Using Versatile All-in-One piggyBac Transposons. Methods Mol Biol. 2015.
. SS18-SSX, the Oncogenic Fusion Protein in Synovial Sarcoma, Is a Cellular Context-Dependent Epigenetic Modifier. PLoS One. 2015;10(11):e0142991.
Efficient and Reproducible Myogenic Differentiation from Human iPS Cells: Prospects for Modeling Miyoshi Myopathy In Vitro. PLoS One. 2013;8(4):e61540.
Transient maternal IL-6 mediates long-lasting changes in neural stem cell pools by deregulating an endogenous self-renewal pathway. Cell Stem Cell. 2013;13(5):564-76.