Biblio

Author Title Type [ Year(Asc)]
Filters: Author is Niwa, Akira  [Clear All Filters]
2023
Kase N, Kitagawa Y, Ikenaka A, Niwa A, Saito MK. A concise model for evaluating interactions between macrophage and skeletal muscle cells during muscle regeneration. Front Cell Dev Biol. 2023;11:1022081.
Saito MK, Osawa M, Tsuchida N, Shiraishi K, Niwa A, Woltjen K, Asaka I, Ogata K, Ito S, Kobayashi S, et al. A disease-specific iPS cell resource for studying rare and intractable diseases. Inflamm Regen. 2023;43(1):43.
Hayashi T, Yano N, Kora K, Yokoyama A, Maizuru K, Kayaki T, Nishikawa K, Osawa M, Niwa A, Takenouchi T, et al. Involvement of mTOR pathway in neurodegeneration in NSF-related developmental and epileptic encephalopathy. Hum Mol Genet. 2023.
Ikenaka A, Kitagawa Y, Yoshida M, Lin C-Y, Niwa A, Nakahata T, Saito MK. SMN promotes mitochondrial metabolic maturation during myogenesis by regulating the MYOD-miRNA axis. Life Sci Alliance. 2023;6(3).
2022
Niwa A, Saito MK. Induction of Human Natural Killer Cells Under Defined Conditions by Seamless Transition from Maintenance Culture of Pluripotent Stem Cells. Methods Mol Biol. 2022;2463:47-52.
2021
Seki R, Ohta A, Niwa A, Sugimine Y, Naito H, Nakahata T, Saito MK. Correction: Induced pluripotent stem cell-derived monocytic cell lines from a NOMID patient serve as a screening platform for modulating NLRP3 inflammasome activity. PLoS One. 2021;16(4):e0249807.
Matsuo S, Nishinaka-Arai Y, Kazuki Y, Oshimura M, Nakahata T, Niwa A, Saito MK. Pluripotent stem cell model of early hematopoiesis in Down syndrome reveals quantitative effects of short-form GATA1 protein on lineage specification. PLoS One. 2021;16(3):e0247595.
2020
Kase N, Terashima M, Ohta A, Niwa A, Honda-Ozaki F, Kawasaki Y, Nakahata T, Kanazawa N, Saito MK. Pluripotent stem cell-based screening identifies CUDC-907 as an effective compound for restoring the in vitro phenotype of Nakajo-Nishimura syndrome. Stem Cells Transl Med. 2020.
2016
Kawasaki Y, Oda H, Ito J, Niwa A, Tanaka T, Hijikata A, Seki R, Nagahashi A, Osawa M, Asaka I, et al. Pluripotent cell-based phenotypic dissection identifies a high-frequency somatic NLRC4 mutation as a cause of autoinflammation. Arthritis Rheumatol. 2016.